05-10-2008, 06:18 PM
(This post was last modified: 05-10-2008, 06:29 PM by Bharatvarsh.)
Because certain things can be used by ignorant idiots doesn't mean one has to stop scientific research, let me give you an example, if my caste is found to have an overall IQ of only 90, that doesn't mean the gov't can set up its own reverse discrimination because of my castes low IQ. We deal with individuals not groups and no amount of data will tell you exactly what IQ a person has, for all we know that person might hav an IQ of 130 and is an outlier of my caste.
We will always have morons in the world and if we stop researching certain things there are many things of benefit that are lost, let me give you an example, sickle cell disease has a very high incidence among people of African descent when compared to people of European descent, a century ago science didn't know why this was, and for all we know ignorant KKK types were probably saying that it was Gods way of punishing them, if scientists stopped research into this because of its sensitive nature we would still be in the dark about it, now we know that it has something to do with an adaptive mechanism against another disease Malaria, which has high prevalnce in Africa (if anyone wants to know the full explanation), check:
http://en.wikipedia.org/wiki/Sickle-cell_disease
Or how about Tay-Sachs which was mostly found in Ashkenazi Jews, cystic-fibrosis in people of European descent.
<!--QuoteBegin-->QUOTE<!--QuoteEBegin-->Historical significance
Tay-Sachs disease has become a model for the prevention of all genetic diseases. In the United States before 1970, the disease affected about 50â70 infants each year in Ashkenazi Jewish families. About 10 cases occurred each year in infants from families without identifiable risk factors. Before 1970, the disease had never been diagnosed at the time of birth. Physicians saw the disease for the first time in infants that failed to thrive, and they could do nothing for the parents or family. Although the genetic basis of the disease was understood, antenatal testing was not available, and families with a Tay-Sachs infant faced a one in four probability of another devastating outcome with each future pregnancy.[5]
Michael Kaback, a medical resident in pediatric neurology at Johns Hopkins University, saw two Tay-Sachs families in 1969. At the time, researchers had just uncovered the biochemical basis of the disease as the failure of an enzyme in a critical metabolic pathway. Kaback developed and later automated an enzyme assay test for detecting heterozygotes (carriers). This inexpensive test proved statistically reliable, with low rates of both errors and false positives. For the first time in medical history, it was possible to screen broadly for a genetic disease, and a physician or medical professional could counsel a family on strategies for prevention. Within a few decades, the disease had been virtually eliminated among Ashkenazi Jews. Most cases today are in families that do not have identifiable risk factors.[5]
Kaback and his associates also developed the first mass screening program for genetic disease. Every aspect of this landmark study was meticulously planned, including community liaison, blood-draw procedure, laboratory set-up, assay protocol, and follow-up genetic counseling. On a Sunday in May 1971, more than 1800 young adults of Ashkenazi Jewish ancestry in the Baltimore and Washington D.C. area were voluntarily screened for carrier status.[35] The success of the program demonstrated the efficacy of voluntary screening of an identifiable at-risk populations. Within a few years, these screening programs had been repeated among Ashkenazi Jews throughout the United States, Canada, western Europe, and Israel.[36][37][38]
In the first 30 years of testing, from 1969 through 1998, more than 1.3 million persons were tested, and 48,864 carriers were identified.
http://en.wikipedia.org/wiki/Tay-Sachs_disease<!--QuoteEnd--><!--QuoteEEnd-->
If scientists instead said that we are all equally prone to the disease just to keep the PC crowd happy, people would probably still be suffering from the disease.
In the case of Jews, I am 100% sure that some nutty xtian retard had said that its Gods way of punishing them for killing the mythical Jesus.
Or how about this drug that went on clinical trial most recently specifically for people of African descent:
http://www.washingtonpost.com/ac2/wp-dyn/A...anguage=printer
Finally can anyone honestly tell me that even with state of the art diet and health care the Mbuti will produce NBA stars:
http://en.wikipedia.org/wiki/Mbuti
Their average height is 137cm or 4'6, which is too low to be caused solely through poor diet (even medieval Europeans or Indians were taller).
We will always have morons in the world and if we stop researching certain things there are many things of benefit that are lost, let me give you an example, sickle cell disease has a very high incidence among people of African descent when compared to people of European descent, a century ago science didn't know why this was, and for all we know ignorant KKK types were probably saying that it was Gods way of punishing them, if scientists stopped research into this because of its sensitive nature we would still be in the dark about it, now we know that it has something to do with an adaptive mechanism against another disease Malaria, which has high prevalnce in Africa (if anyone wants to know the full explanation), check:
http://en.wikipedia.org/wiki/Sickle-cell_disease
Or how about Tay-Sachs which was mostly found in Ashkenazi Jews, cystic-fibrosis in people of European descent.
<!--QuoteBegin-->QUOTE<!--QuoteEBegin-->Historical significance
Tay-Sachs disease has become a model for the prevention of all genetic diseases. In the United States before 1970, the disease affected about 50â70 infants each year in Ashkenazi Jewish families. About 10 cases occurred each year in infants from families without identifiable risk factors. Before 1970, the disease had never been diagnosed at the time of birth. Physicians saw the disease for the first time in infants that failed to thrive, and they could do nothing for the parents or family. Although the genetic basis of the disease was understood, antenatal testing was not available, and families with a Tay-Sachs infant faced a one in four probability of another devastating outcome with each future pregnancy.[5]
Michael Kaback, a medical resident in pediatric neurology at Johns Hopkins University, saw two Tay-Sachs families in 1969. At the time, researchers had just uncovered the biochemical basis of the disease as the failure of an enzyme in a critical metabolic pathway. Kaback developed and later automated an enzyme assay test for detecting heterozygotes (carriers). This inexpensive test proved statistically reliable, with low rates of both errors and false positives. For the first time in medical history, it was possible to screen broadly for a genetic disease, and a physician or medical professional could counsel a family on strategies for prevention. Within a few decades, the disease had been virtually eliminated among Ashkenazi Jews. Most cases today are in families that do not have identifiable risk factors.[5]
Kaback and his associates also developed the first mass screening program for genetic disease. Every aspect of this landmark study was meticulously planned, including community liaison, blood-draw procedure, laboratory set-up, assay protocol, and follow-up genetic counseling. On a Sunday in May 1971, more than 1800 young adults of Ashkenazi Jewish ancestry in the Baltimore and Washington D.C. area were voluntarily screened for carrier status.[35] The success of the program demonstrated the efficacy of voluntary screening of an identifiable at-risk populations. Within a few years, these screening programs had been repeated among Ashkenazi Jews throughout the United States, Canada, western Europe, and Israel.[36][37][38]
In the first 30 years of testing, from 1969 through 1998, more than 1.3 million persons were tested, and 48,864 carriers were identified.
http://en.wikipedia.org/wiki/Tay-Sachs_disease<!--QuoteEnd--><!--QuoteEEnd-->
If scientists instead said that we are all equally prone to the disease just to keep the PC crowd happy, people would probably still be suffering from the disease.
In the case of Jews, I am 100% sure that some nutty xtian retard had said that its Gods way of punishing them for killing the mythical Jesus.
Or how about this drug that went on clinical trial most recently specifically for people of African descent:
http://www.washingtonpost.com/ac2/wp-dyn/A...anguage=printer
Finally can anyone honestly tell me that even with state of the art diet and health care the Mbuti will produce NBA stars:
http://en.wikipedia.org/wiki/Mbuti
Their average height is 137cm or 4'6, which is too low to be caused solely through poor diet (even medieval Europeans or Indians were taller).